Treatment of dividing cells with a low dose of the antifungal drug benomyl, which destabilizes microtubules, slows down correct spindle assembly. but at such doses, the spindle is eventually formed and the cells survive. however, mutations in some genes confer benomyl sensitivity: the mutant cells die because they fail to arrest the cell cycle in the presence of unattached kinetochores and progress through anaphase, with disastrous consequences. which of the following would you expect to be a benomyl-sensitive mutant? a. a loss-of-function mutant in the gene encoding mad2.
b. a mutation causing the overexpression of cdc20.
c. a loss-of-function mutation in the gene encoding a kinase that inhibits cdc20–apc/c.
d. a loss-of-function mutation in the gene encoding a tubulin subunit.
e. all of the above.

The answer is letter A.

Explanation:

A loss-of-function mutant in the gene encoding Mad2.

well we can't tell you because we cant see the time give a screen shot or something.

both anaerobic and aerobic respiration use glucose (or other carbohydrates), proteins and lipids to produce atp and nadh. cell uses those energy molecules, obtained from food and   breaks them down during the chemical reactions of respiration. in anaerobic respiration, nadh is regenerated to nad+ by enzymes lactate dehydrogenase (lactate is produced, e.g. in muscle cells) or by alcohol dehydrogenase where ethanol is produced (alcohol fermentation), while in aerobic respiration, electrons from nadh are deposited in the mitochondria to regenerate nad+ (electron transport chain. anaerobic respiration produces 2 atp per molecule of glucose compared to aerobic respiration where about 38 atp molecules are produced.