In a series of experiments, genes that code for mutant forms of a receptor tyrosine kinase (rtk) are introduced into cells. the cells also can express their own normal form of the receptor from their normal gene, but the mutant genes are constructed so that the mutant rtk is expressed at considerably higher concentration than the normal rtk. what would be the consequences of introducing a mutant gene that codes for an rtk

a) lacking its extracellular domain?

b) lacking its intracellular domain?

the answer is otpion c) understanding

he wants to be heard one more time. he wants to be understood. he needs the truth one more time.

the answer is: the „power stroke“ step.

muscle contraction can be divided into a few steps:

1. the release of ach (acetylcholine) in the synaptic cleft, binding of ach for its receptor and consequently opening of the ca channels.

2. the release of ca, binding of ca for the troponin

3. troponin shifting the tropomyosin, which was blocking the active site on the actin

4. myosin heads attaching to actin by breaking down atp to adp+ pi

5.   formation of the ‘cross-bridge’ on the active site of the actin filament by myosin head

6. “power stroke” -the cross bridge pulls actin, which slides over the myosin

9.     7. the release of adp completes the cross-bridge movement and atp attaches to myosin, breaking the actin-myosin cross bridge.

answerglucose;