How is the dispersive model different from the semiconservative model of DNA replication? 1. In the dispersive model, each strand of both daughter molecules contains a mixture of old and newly synthesized DNA, and in the semiconservative model, each new double helix consists of one old and one new strand.
2. In the dispersive model, the old double helix is degraded, and in the semiconservative model half of its nucleotides are used in the construction of two new double helices.
3. In the dispersive model, one of the two resulting double helices is made of two old strands, and in the semiconservative model the other is made of two new strands.
4. In the dispersive model, one strand of the new double helix is made of DNA, and in the semiconservative model the other strand is made of RNA.

With the description of the different cell walls, membranes, and associated proteins set in the students' minds, you now need to introduce them to the idea that the cell wall can also act as a foundation to build things upon. bacterial appendages require a strong foundation that will offer the support needed to move and function in a dynamic world. for example, flagella are long, whiplike protein structures that are used by many gram-positive and gram-negative bacteria for locomotion. in order to function effectively, a flagellum must be firmly anchored to the cell wall. how will you be able to get across the idea that the peptidoglycan cell wall is strong enough to support such a mechanism? with a protein rod that passes through the cell wall and protein rings used to anchor it in the membranes, these basal bodies are the rudimentary biological motors that use atp power to spin the hook and the flagella attached to it. bacterial flagella have a biological motor that spins within the cell wall and is powered by atp. this allows the flagella to spin in a whiplike motion to propel the bacterium.

How does the author's purpose in this passage differ from an author writing about her personal struggle living with asthma?

In 1959, doctors began using the powerful antibiotic methicillin to treat infections of staphylococcus aureus, but within two years, methicillin-resistant strains of s. aureus (mrsa) appeared. how did the resistant strains of s. aureus emerge? in 1959, doctors began using the powerful antibiotic methicillin to treat infections of staphylococcus aureus, but within two years, methicillin-resistant strains of s. aureus (mrsa) appeared. how did the resistant strains of s. aureus emerge? staphylococcus aureus bacteria that were able to synthesize cell walls using a protein that was not affected by methicillin survived the methicillin treatments and reproduced at higher rates than did other individuals. over time, these resistant individuals became increasingly common. in response to treatment of staphylococcus aureus infections with methicillin, some bacteria began to synthesize cell walls using a protein that was not affected by methicillin. these bacteria survived the methicillin treatments and reproduced at higher rates than did other individuals. over time, these resistant individuals became increasingly common. in response to treatment of staphylococcus aureus infections with methicillin, bacterial populations gradually began to synthesize cell walls using a protein that was not affected by methicillin.