Why do second-generation h1 blockers cause less sedation then first-generation h1 blockers? they are less potent

Conrad jackson is a 28-year-old man who presents to the emergency department with severe fatigue and dehydration secondary to a 4-day history of vomiting. during the interview, he describes attending a family reunion and states that perhaps he "ate something bad". upon admission his vital signs are a temperature of 102.7 f, heart rate of 116 bpm, respiratory rate of 18 breaths/min, and blood pressure of 86/54 mm hg. the nurse also notes the patient has dry mucuos membranes and tenting of skin. the physician orders an iv to be started with 0.45% normal saline, and orders a serum electrolytes and an arterial blood gas. the following results are returned from the laboratory: sodium (na+) 150 potassium (k+) 5.5 chloride (cl-) 110 bun 42 creatinine 0.8 glucose 86 ph 7.32 paco2 35 hco3 20 pao2 90 o2sat 98% a. what is your interpretation of this arterial blood gas sample? b. explain the high potassium in this patient. c. calculate the patient's anion gap: d. what is the interpretation of this anion gap?